Resident macrophages keep mitochondria running in the heart
The high workload of cardiac myocytes,coupled with their negligible turnover,requires that compensatory mechanisms act in the heart to address ongoing cellular damage and extruded organelle components that would otherwise accu?mulate.A recent study in Cell by Nicolás-ávila et al.illustrates how cardiac-resident macrophages serve such a role,by clearing extruded mitochondrial debris to preserve organ homeostasis and maintain optimal cardiac function.
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2021-01-29(万方平台首次上网日期,不代表论文的发表时间)
共2页
1057-1058