Tumor suppressor ASXL1 is essential for the activation of INK4B expression in response to oncogene activity and anti-proliferative signals
25
The work in the Helin lab was supported by grants from the Excellence Program of the University of Copenhagen, the Danish Cancer Society, the Novo Nordisk Foundation, and the European Commission”s 7th Framework Program 4DCellFate 277899.The work in the Helin and the Jensen labs was supported by the Danish National Research Foundation DNRF82.The work in the Rappsilber lab was supported by Wellcome Trust 103139, 092076, and 091020.The Project is sponsored by the Scientific Research Foundation for the Returned Overseas Chinese Scholars, State Education Ministry.The work also received support from the National Natural Science Foundation of China 81470295 to ZX, 81171899 to Xi Wang, 31570774 to Xudong Wu and 81170007 to GX.
2015-12-10(万方平台首次上网日期,不代表论文的发表时间)
1205-1218