Preferential loss of mismatch repair function in refractory and relapsed acute myeloid leukemia: potential contribution to AML progression
acute myeloid leukemia、molecular mechanisms、complete remission、mismatch repair、control samples、amino acid
18
Q2(细胞生物学)
supported by the National Institute of HealthCA104333;American Cancer Society FundIRG-163H
2008-05-28(万方平台首次上网日期,不代表论文的发表时间)
共9页
281-289